Patients | BreathLab

Resources For Patients

Dysfunctional Breathing

Accurate assessment and diagnosis can prevent excessive use of drug therapy and result in the effective management of the cause of the individual’s complaint through cost-effective approaches such as reassurance, advice, breathing retraining, and vocal exercises.

Blood PH

Respiratory alkalosis occurs when you breathe too fast or too deep and carbon dioxide levels drop too low. This causes the pH of the blood to rise and become too alkaline.

Hyperventilation is typically the underlying cause of respiratory alkalosis. Hyperventilation is also known as over-breathing. Someone who is hyperventilating breathes very deeply or rapidly.

Mouth Breathing and Caries

Caries has been placed in correlation with the entry of air through the mouth (associated with the surface dehydration and disappearance of the film of saliva from the tooth surface); the regular intake of fermentable carbohydrates in the diet involves lowering the pH, resulting in the formation of plaque mostly composed of acid-forming and acid-tolerable species, such as Streptococcus mutans (S. mutans) and Lactobacilli

Respiratory Alkalosis

result from over-breathing

The most frequent acid-base disturbance encountered in clinical practice.

Although this acid-base disturbance often is considered benign, evidence suggests that the alkalemia of primary hypocapnia can cause clinically significant decreases in tissue oxygen delivery.

Mild respiratory alkalosis often serves as a marker of an underlying disease and may not require therapeutic intervention. In contrast, severe respiratory alkalosis should be approached with a sense of urgency and be aggressively corrected.

O2 in Blood

ARTERIAL OXYGEN CONTENT IN THE REGULATION OF CEREBRAL BLOOD

A highly significant relationship was found between CBF and arterial oxygen content, CBF altering to maintain the rate of transport of oxygen to the brain within certain limits.

Reduced Oxidative Stress and Blood Lactic Acidosis

In divers, the changes in lactic acid, TBARS, RAA, and GSH concentrations were markedly reduced after static and dynamic apnea, as well as after control exercise. Thus, human subjects involved in a long duration training program of breath-hold diving have reduced post-apnea as well as post-exercise blood acidosis and oxidative stress, mimicking the responses of diving animals.